Hemin-Dependent Modulation of the Lipid A Structure of Porphyromonas gingivalis Lipopolysaccharide | Infection and Immunity
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis
Circular RNA PPP1CC promotes Porphyromonas gingivalis-lipopolysaccharide-induced pyroptosis of vascular smooth muscle cells by activating the HMGB1/TLR9/AIM2 pathway - Jie Liu, Yong Wang, Yaoyun Liao, Ying Zhou, Jijin Zhu, 2021
GI Bacteria Affects Rheumatoid Arthritis Disease Markers | Chondrex, Inc.
Molecules | Free Full-Text | Porphyromonas gingivalis Lipopolysaccharide Induced Proliferation and Activation of Natural Killer Cells in Vivo
Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activatio
Porphyromonas Gingivalis in the Pathogenesis of Alzheimer's Disease and Its Therapeutic Target
InvivoGen] LPS-PG (Standard) : DAWINBIO
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
Role of TLR4 signaling on Porphyromonas gingivalis LPS-induced cardiac dysfunction in mice | PLOS ONE
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
The Effect of Porphyromonas gingivalis Lipopolysaccharide on the Pyroptosis of Gingival Fibroblasts | SpringerLink
Lipoxin A4 promotes autophagy and inhibits overactivation of macrophage inflammasome activity induced by Pg LPS - Jie Zhao, Wenjing Geng, Kefei Wan, Kailei Guo, Fengjun Xi, Xiangqun Xu, Xiujuan Xiong, Xu Huang,
Visfatin regulates Pg LPS-induced proinflammatory/prodegradative effects in healthy and inflammatory periodontal cells partially via NF-κB pathway - ScienceDirect
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
Porphyromonas gingivalis Outer Membrane Vesicles as the Major Driver of and Explanation for Neuropathogenesis, the Cholinergic Hypothesis, Iron Dyshomeostasis, and Salivary Lactoferrin in Alzheimer's Disease - IOS Press
In Vivo Up-Regulation of Kinin B1 Receptors after Treatment with Porphyromonas gingivalis Lipopolysaccharide in Rat Paw | Journal of Pharmacology and Experimental Therapeutics
IJMS | Free Full-Text | P. gingivalis-LPS Induces Mitochondrial Dysfunction Mediated by Neuroinflammation through Oxidative Stress
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
em>Porphyromonas gingivalis</em> lipopolysaccharide enhances the proliferation of human periodontal ligament cells via upregulation of cyclin D1, cyclin A and cyclin B1
LPS-PG | Lipopolysaccharide from Porphyromonas Gingivalis